Anabolic steroids metabolism, anabolic steroids benefits
Anabolic steroids metabolism
In rats, anabolic steroids also act in the peripheral metabolism of thyroid hormones and seem to exert an important proliferative effect on thyroid cellsin both animal models of congenital hypothyroidism  ,  -  and humans  . A study by Dangour et al. found that chronic administration of a mixed steroid (testosterone, nandrolone, nandrolone decanoate) to rats increased the concentrations of free thyroxine in the plasma of the rats and the number of thyroxine-bound thyroxine receptors in the rat thyroid glands, which could result in the up-scaling of steroid hormones by their action in the rat. Although a possible direct action (by acting in the mitochondria) of steroid hormones on the thyroid cell is not well documented, it is plausible, metabolism anabolic steroids. Recently, Chen et al. showed that a mixed steroid (testosterone, nandrolone, and nandrolone decanoate) administered chronically to rats increased thyroid hormone content in the serum and the cells of the thyroid, which could result in the up-scaling of thyroid hormone to normal levels by its action in the endoplasmic reticulum  . Additionally, by altering the expression of the transcription factors thyroid hormone dependent growth factor 1 (TRGFRF1) and TGF-β 1 (TRGFRF2) in rat thyroid glands, a recent study showed that these effects were mediated by steroids, anabolic steroids metabolism. In addition, by using a transgenic mouse model of congenital hypothyroidism (SCID), Hensley et al, anabolic-androgenic steroids. found that chronic administration of anabolic steroids in these animals significantly decreased expression of the thyroid-stimulating hormone receptor mRNA in the hypothalamus and the rat brain and increased thyroid hormone levels in the brain and the pituitary  ,  , anabolic-androgenic steroids. In the nucleus, an anabolic steroid receptor subunit (AR) gene was found increased in rat thyroid gland, which can be inhibited by the selective estrogen receptor antagonist drosogostat  , (a steroid hormone receptor blocker)  and the synthetic analogs of the steroid hormone-like 2 (SYN) and 3 (TRPA1) (all steroids)  ,  . As in the rat, acute administration of a synthetic analog of anabolic steroids (prostaglandin E2) or dexamethasone (a steroid hormone receptor blocker) abolished these effects of anabolic steroids on pituitary adrenal glands.
Anabolic steroids benefits
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